2020
|
Balbino, B; Herviou, P; o Godon,; Stackowicz, J; Goff, O R; Iannascoli, B; Sterlin, D; Brûlé, S; Millot, G A; Harris, F M; Voronina, V A; Nadeau, K C; Macdonald, L E; Murphy, A J; Bruhns, P; Reber, L L The anti-IgE mAb omalizumab induces adverse reactions by engaging Fcγ receptors. Journal Article J Clin Invest, 130 (3), pp. 1330-1335, 2020. Links | BibTeX @article{B2020,
title = {The anti-IgE mAb omalizumab induces adverse reactions by engaging Fcγ receptors.},
author = {B Balbino and P Herviou and o Godon and J Stackowicz and O R Goff and B Iannascoli and D Sterlin and S Brûlé and G A Millot and F M Harris and V A Voronina and K C Nadeau and L E Macdonald and A J Murphy and P Bruhns and L L Reber},
url = {https://pubmed-ncbi-nlm-nih-gov.proxy.insermbiblio.inist.fr/31770111/},
doi = {10.1172/JCI129697},
year = {2020},
date = {2020-03-02},
journal = {J Clin Invest},
volume = {130},
number = {3},
pages = {1330-1335},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
|
2019
|
Serhan, N; Basso, L; Sibilano, R; Petitfils, C; Meixiong, J; Bonnart, C; Reber, L L; Marichal, T; Starkl, P; Cenac, N; Dong, X; Tsai, M; Galli, S J; Gaudenzio, N House dust mites activate nociceptor-mast cell clusters to drive type 2 skin inflammation Journal Article Nat Immunol, 20 (11), pp. 1435-1443, 2019. Links | BibTeX @article{N2019,
title = {House dust mites activate nociceptor-mast cell clusters to drive type 2 skin inflammation},
author = {Serhan, N. and Basso, L. and Sibilano, R. and Petitfils, C. and Meixiong, J. and Bonnart, C. and Reber, L L. and Marichal, T. and Starkl, P. and Cenac, N. and Dong, X. and Tsai, M. and Galli, S J. and Gaudenzio, N.},
url = {https://pubmed-ncbi-nlm-nih-gov.proxy.insermbiblio.inist.fr/31591569/},
doi = {10.1038/s41590-019-0493-z},
year = {2019},
date = {2019-11-20},
journal = {Nat Immunol},
volume = {20},
number = {11},
pages = {1435-1443},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
|
Adoue, V; Binet, B; Malbec, A; Fourquet, J; Romagnoli, P; van Meerwijk, J P M; Amigorena, S; Joffre, O P The Histone Methyltransferase SETDB1 Controls T Helper Cell Lineage Integrity by Repressing Endogenous Retroviruses. Journal Article Immunity, 50 , pp. 629–644, 2019. Abstract | Links | BibTeX @article{Adoue2019,
title = {The Histone Methyltransferase SETDB1 Controls T Helper Cell Lineage Integrity by Repressing Endogenous Retroviruses.},
author = {Adoue, V and Binet, B and Malbec, A and Fourquet, J and Romagnoli, P and van Meerwijk, J P M and Amigorena, S and Joffre, O P},
url = {https://linkinghub.elsevier.com/retrieve/pii/S1074761319300032},
doi = {10.1016/j.immuni.2019.01.003},
year = {2019},
date = {2019-01-01},
journal = {Immunity},
volume = {50},
pages = {629--644},
abstract = {Upon activation, naive CD4+ T cells differentiate into distinct T cell subsets via processes reliant on epigenetically regulated, lineage-specific developmental programs. Here, we examined the function of the histone methyltransferase SETDB1 in T helper (Th) cell differentiation. Setdb1-/- naive CD4+ T cells exhibited exacerbated Th1 priming, and when exposed to a Th1-instructive signal, Setdb1-/- Th2 cells crossed lineage boundaries and acquired a Th1 phenotype. SETDB1 did not directly control Th1 gene promoter activity but relied instead on deposition of the repressive H3K9me3 mark at a restricted and cell-type-specific set of endogenous retroviruses (ERVs) located in the vicinity of genes involved in immune processes. Refined bioinformatic analyses suggest that these retrotransposons regulate Th1 gene cis-regulatory elements or act as Th1 gene enhancers. Thus, H3K9me3 deposition by SETDB1 ensures Th cell lineage integrity by repressing a repertoire of ERVs that have been exapted into cis-regulatory modules to shape and control the Th1 gene network.},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
Upon activation, naive CD4+ T cells differentiate into distinct T cell subsets via processes reliant on epigenetically regulated, lineage-specific developmental programs. Here, we examined the function of the histone methyltransferase SETDB1 in T helper (Th) cell differentiation. Setdb1-/- naive CD4+ T cells exhibited exacerbated Th1 priming, and when exposed to a Th1-instructive signal, Setdb1-/- Th2 cells crossed lineage boundaries and acquired a Th1 phenotype. SETDB1 did not directly control Th1 gene promoter activity but relied instead on deposition of the repressive H3K9me3 mark at a restricted and cell-type-specific set of endogenous retroviruses (ERVs) located in the vicinity of genes involved in immune processes. Refined bioinformatic analyses suggest that these retrotransposons regulate Th1 gene cis-regulatory elements or act as Th1 gene enhancers. Thus, H3K9me3 deposition by SETDB1 ensures Th cell lineage integrity by repressing a repertoire of ERVs that have been exapted into cis-regulatory modules to shape and control the Th1 gene network. |
Domingo, Cristina ; Fraissinet, Juliane ; Ansah, Patrick O; Kelly, Corey ; Bhat, Niranjan ; Sow, Samba O; Mejía, José E Long-term immunity against yellow fever in children vaccinated during infancy: a longitudinal cohort study Journal Article Lancet Infect Dis, 2019, ISSN: 1473-3099. Abstract | Links | BibTeX @article{RN3090,
title = {Long-term immunity against yellow fever in children vaccinated during infancy: a longitudinal cohort study},
author = {Domingo, Cristina and Fraissinet, Juliane and Ansah, Patrick O. and Kelly, Corey and Bhat, Niranjan and Sow, Samba O. and Mejía, José E.},
doi = {10.1016/S1473-3099(19)30323-8},
issn = {1473-3099},
year = {2019},
date = {2019-01-01},
journal = {Lancet Infect Dis},
abstract = {Background. A single dose of vaccine against yellow fever is routinely administered to infants aged 9–12 months under the Expanded Programme on Immunization, but the long-term outcome of vaccination in this age group is unknown. We aimed to evaluate the long-term persistence of neutralising antibodies to yellow fever virus following routine vaccination in infancy.
Methods. We did a longitudinal cohort study, using a microneutralisation assay to measure protective antibodies against yellow fever in Malian and Ghanaian children vaccinated around age 9 months and followed up for 4·5 years (Mali), or 2·3 and 6·0 years (Ghana). Healthy children with available day-0 sera, a complete follow-up history, and no record of yellow fever revaccination were included; children seropositive for yellow fever at baseline were excluded. We standardised antibody concentrations with reference to the yellow fever WHO International Standard.
Findings. We included 587 Malian and 436 Ghanaian children vaccinated between June 5, 2009, and Dec 26, 2012. In the Malian group, 296 (50·4%, 95% CI 46·4–54·5) were seropositive (antibody concentration ≥0·5 IU/mL) 4·5 years after vaccination. Among the Ghanaian children, 121 (27·8%, 23·5–32·0) were seropositive after 2·3 years. These results show a large decrease from the proportions of seropositive infants 28 days after vaccination, 96·7% in Mali and 72·7% in Ghana, reported by a previous study of both study populations. The number of seropositive children increased to 188 (43·1%, 95% CI 38·5–47·8) in the Ghanaian group 6·0 years after vaccination, but this result might be confounded by unrecorded revaccination or natural infection with wild yellow fever virus during a 2011–12 outbreak in northern Ghana.
Interpretation. Rapid waning of immunity during the early years after vaccination of 9-month-old infants argues for a revision of the single-dose recommendation for this target population in endemic countries. The short duration of immunity in many vaccinees suggests that booster vaccination is necessary to meet the 80% population immunity threshold for prevention of yellow fever outbreaks.
},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
Background. A single dose of vaccine against yellow fever is routinely administered to infants aged 9–12 months under the Expanded Programme on Immunization, but the long-term outcome of vaccination in this age group is unknown. We aimed to evaluate the long-term persistence of neutralising antibodies to yellow fever virus following routine vaccination in infancy.
Methods. We did a longitudinal cohort study, using a microneutralisation assay to measure protective antibodies against yellow fever in Malian and Ghanaian children vaccinated around age 9 months and followed up for 4·5 years (Mali), or 2·3 and 6·0 years (Ghana). Healthy children with available day-0 sera, a complete follow-up history, and no record of yellow fever revaccination were included; children seropositive for yellow fever at baseline were excluded. We standardised antibody concentrations with reference to the yellow fever WHO International Standard.
Findings. We included 587 Malian and 436 Ghanaian children vaccinated between June 5, 2009, and Dec 26, 2012. In the Malian group, 296 (50·4%, 95% CI 46·4–54·5) were seropositive (antibody concentration ≥0·5 IU/mL) 4·5 years after vaccination. Among the Ghanaian children, 121 (27·8%, 23·5–32·0) were seropositive after 2·3 years. These results show a large decrease from the proportions of seropositive infants 28 days after vaccination, 96·7% in Mali and 72·7% in Ghana, reported by a previous study of both study populations. The number of seropositive children increased to 188 (43·1%, 95% CI 38·5–47·8) in the Ghanaian group 6·0 years after vaccination, but this result might be confounded by unrecorded revaccination or natural infection with wild yellow fever virus during a 2011–12 outbreak in northern Ghana.
Interpretation. Rapid waning of immunity during the early years after vaccination of 9-month-old infants argues for a revision of the single-dose recommendation for this target population in endemic countries. The short duration of immunity in many vaccinees suggests that booster vaccination is necessary to meet the 80% population immunity threshold for prevention of yellow fever outbreaks.
|
Piliponsky, A M; Shubin, N J; Lahiri, A K; Truong, P; Clauson, M; Niino, K; Tsuha, A L; Nedospasov, S A; Karasuyama, H; Reber, L L; Tsai, M; Mukai, K; Galli, S J Basophil-derived tumor necrosis factor can enhance survival in a sepsis model in mice Journal Article Nat Immunol, 20 (2), pp. 129-140, 2019, ISSN: 1529-2916 (Electronic) 1529-2908 (Linking). Links | BibTeX @article{RN1b,
title = {Basophil-derived tumor necrosis factor can enhance survival in a sepsis model in mice},
author = {Piliponsky, A. M. and Shubin, N. J. and Lahiri, A. K. and Truong, P. and Clauson, M. and Niino, K. and Tsuha, A. L. and Nedospasov, S. A. and Karasuyama, H. and Reber, L. L. and Tsai, M. and Mukai, K. and Galli, S. J.},
url = {https://www.ncbi.nlm.nih.gov/pubmed/30664762},
doi = {10.1038/s41590-018-0288-7},
issn = {1529-2916 (Electronic) 1529-2908 (Linking)},
year = {2019},
date = {2019-01-01},
journal = {Nat Immunol},
volume = {20},
number = {2},
pages = {129-140},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
|
2018
|
Rosa, N; Triffaux, E; Robert, V; Mars, M; Klein, M; Bouchaud, G; Canivet, A; Magnan, A; Guery, J C; Pelletier, L; Savignac, M The beta and alpha2delta auxiliary subunits of voltage-gated calcium channel 1 (Cav1) are required for TH2 lymphocyte function and acute allergic airway inflammation Journal Article J Allergy Clin Immunol, 142 (3), pp. 892-903, 2018, ISSN: 1097-6825 (Electronic) 0091-6749 (Linking). Links | BibTeX @article{RN24,
title = {The beta and alpha2delta auxiliary subunits of voltage-gated calcium channel 1 (Cav1) are required for TH2 lymphocyte function and acute allergic airway inflammation},
author = {Rosa, N. and Triffaux, E. and Robert, V. and Mars, M. and Klein, M. and Bouchaud, G. and Canivet, A. and Magnan, A. and Guery, J. C. and Pelletier, L. and Savignac, M.},
url = {https://www.ncbi.nlm.nih.gov/pubmed/29129580
https://www.sciencedirect.com/science/article/pii/S0091674917317402},
doi = {10.1016/j.jaci.2017.09.045},
issn = {1097-6825 (Electronic) 0091-6749 (Linking)},
year = {2018},
date = {2018-09-01},
journal = {J Allergy Clin Immunol},
volume = {142},
number = {3},
pages = {892-903},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
|
Souyris, M; Cenac, C; Azar, P; Daviaud, D; Canivet, A; Grunenwald, S; Pienkowski, C; Chaumeil, J; Mejia, J E; Guery, J C TLR7 escapes X chromosome inactivation in immune cells Journal Article Sci Immunol, 3 (19), 2018, ISSN: 2470-9468 (Electronic) 2470-9468 (Linking), (In the top 5% of all research outputs scored by Altmetric. http://www.altmetric.com/details/32261033). Links | BibTeX @article{RN25b,
title = {TLR7 escapes X chromosome inactivation in immune cells},
author = {Souyris, M. and Cenac, C. and Azar, P. and Daviaud, D. and Canivet, A. and Grunenwald, S. and Pienkowski, C. and Chaumeil, J. and Mejia, J. E. and Guery, J. C.},
url = {https://www.ncbi.nlm.nih.gov/pubmed/29374079},
doi = {10.1126/sciimmunol.aap8855},
issn = {2470-9468 (Electronic) 2470-9468 (Linking)},
year = {2018},
date = {2018-01-26},
journal = {Sci Immunol},
volume = {3},
number = {19},
note = {In the top 5% of all research outputs scored by Altmetric. http://www.altmetric.com/details/32261033},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
|
Turner, M; Diaz-Munoz, M D RNA-binding proteins control gene expression and cell fate in the immune system Journal Article Nat Immunol, 19 (2), pp. 120-129, 2018, ISSN: 1529-2916 (Electronic)
1529-2908 (Linking). Links | BibTeX @article{RN1b,
title = {RNA-binding proteins control gene expression and cell fate in the immune system},
author = {Turner, M. and Diaz-Munoz, M. D.},
url = {https://www.ncbi.nlm.nih.gov/pubmed/29348497},
doi = {10.1038/s41590-017-0028-4},
issn = {1529-2916 (Electronic)
1529-2908 (Linking)},
year = {2018},
date = {2018-01-01},
journal = {Nat Immunol},
volume = {19},
number = {2},
pages = {120-129},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
|
Eissmann, M F; Dijkstra, C; Wouters, M A; Baloyan, D; Mouradov, D; Nguyen, P M; Davalos-Salas, M; Putoczki, T L; Sieber, O M; Mariadason, J M; Ernst, M; Masson, F Interleukin 33 Signaling Restrains Sporadic Colon Cancer in an Interferon-gamma-Dependent Manner Journal Article Cancer Immunol Res, 6 (4), pp. 409-421, 2018, ISSN: 2326-6074 (Electronic) 2326-6066 (Linking). Links | BibTeX @article{RN1b,
title = {Interleukin 33 Signaling Restrains Sporadic Colon Cancer in an Interferon-gamma-Dependent Manner},
author = {Eissmann, M. F. and Dijkstra, C. and Wouters, M. A. and Baloyan, D. and Mouradov, D. and Nguyen, P. M. and Davalos-Salas, M. and Putoczki, T. L. and Sieber, O. M. and Mariadason, J. M. and Ernst, M. and Masson, F.},
url = {https://www.ncbi.nlm.nih.gov/pubmed/29463593},
doi = {10.1158/2326-6066.CIR-17-0218},
issn = {2326-6074 (Electronic) 2326-6066 (Linking)},
year = {2018},
date = {2018-01-01},
journal = {Cancer Immunol Res},
volume = {6},
number = {4},
pages = {409-421},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
|
Houmadi, R; Guipouy, D; Rey-Barroso, J; Vasconcelos, Z; Cornet, J; Manghi, M; Destainville, N; Valitutti, S; Allart, S; Dupre, L The Wiskott-Aldrich Syndrome Protein Contributes to the Assembly of the LFA-1 Nanocluster Belt at the Lytic Synapse Journal Article Cell Rep, 22 (4), pp. 979-991, 2018, ISSN: 2211-1247 (Electronic). Links | BibTeX @article{RN3b,
title = {The Wiskott-Aldrich Syndrome Protein Contributes to the Assembly of the LFA-1 Nanocluster Belt at the Lytic Synapse},
author = {Houmadi, R. and Guipouy, D. and Rey-Barroso, J. and Vasconcelos, Z. and Cornet, J. and Manghi, M. and Destainville, N. and Valitutti, S. and Allart, S. and Dupre, L.},
url = {https://www.ncbi.nlm.nih.gov/pubmed/29386139},
doi = {10.1016/j.celrep.2017.12.088},
issn = {2211-1247 (Electronic)},
year = {2018},
date = {2018-01-01},
journal = {Cell Rep},
volume = {22},
number = {4},
pages = {979-991},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
|
Gaud, G; Lesourne, R; Love, P E Regulatory mechanisms in T cell receptor signalling Journal Article Nat Rev Immunol, 2018, ISSN: 1474-1741 (Electronic)
1474-1733 (Linking). Links | BibTeX @article{RN1b,
title = {Regulatory mechanisms in T cell receptor signalling},
author = {Gaud, G. and Lesourne, R. and Love, P. E.},
url = {https://www.ncbi.nlm.nih.gov/pubmed/29789755},
doi = {10.1038/s41577-018-0020-8},
issn = {1474-1741 (Electronic)
1474-1733 (Linking)},
year = {2018},
date = {2018-01-01},
journal = {Nat Rev Immunol},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
|
Betourne, A; Szelechowski, M; Thouard, A; Abrial, E; Jean, A; Zaidi, F; Foret, C; Bonnaud, E M; Charlier, C M; Suberbielle, E; Malnou, C E; Granon, S; Rampon, C; Gonzalez-Dunia, D Hippocampal expression of a virus-derived protein impairs memory in mice Journal Article Proc Natl Acad Sci U S A, 115 (7), pp. 1611-1616, 2018, ISSN: 1091-6490 (Electronic) 0027-8424 (Linking). Links | BibTeX @article{RN108,
title = {Hippocampal expression of a virus-derived protein impairs memory in mice},
author = {Betourne, A. and Szelechowski, M. and Thouard, A. and Abrial, E. and Jean, A. and Zaidi, F. and Foret, C. and Bonnaud, E. M. and Charlier, C. M. and Suberbielle, E. and Malnou, C. E. and Granon, S. and Rampon, C. and Gonzalez-Dunia, D.},
url = {https://www.ncbi.nlm.nih.gov/pubmed/29378968},
doi = {10.1073/pnas.1711977115},
issn = {1091-6490 (Electronic) 0027-8424 (Linking)},
year = {2018},
date = {2018-01-01},
journal = {Proc Natl Acad Sci U S A},
volume = {115},
number = {7},
pages = {1611-1616},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
|
Colin, P; Zhou, Z; Staropoli, I; Garcia-Perez, J; Gasser, R; Armani-Tourret, M; Benureau, Y; Gonzalez, N; Jin, J; Connell, B J; Raymond, S; Delobel, P; Izopet, J; Lortat-Jacob, H; Alcami, J; Arenzana-Seisdedos, F; Brelot, A; Lagane, B CCR5 structural plasticity shapes HIV-1 phenotypic properties Journal Article PLoS Pathog, 14 (12), pp. e1007432, 2018, ISSN: 1553-7374 (Electronic) 1553-7366 (Linking). Links | BibTeX @article{RN859,
title = {CCR5 structural plasticity shapes HIV-1 phenotypic properties},
author = {Colin, P. and Zhou, Z. and Staropoli, I. and Garcia-Perez, J. and Gasser, R. and Armani-Tourret, M. and Benureau, Y. and Gonzalez, N. and Jin, J. and Connell, B. J. and Raymond, S. and Delobel, P. and Izopet, J. and Lortat-Jacob, H. and Alcami, J. and Arenzana-Seisdedos, F. and Brelot, A. and Lagane, B.},
url = {https://www.ncbi.nlm.nih.gov/pubmed/30521629},
doi = {10.1371/journal.ppat.1007432},
issn = {1553-7374 (Electronic) 1553-7366 (Linking)},
year = {2018},
date = {2018-01-01},
journal = {PLoS Pathog},
volume = {14},
number = {12},
pages = {e1007432},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
|
2017
|
Tauber, Maïthé; Boulanouar, Kader; Diene, Gwenaelle; Çabal-Berthoumieu, Sophie; Ehlinger, Virginie; Fichaux-Bourin, Pascale; Molinas, Catherine; Faye, Sandy; Valette, Marion; Pourrinet, Jeanne; Cessans, Catie; Viaux-Sauvelon, Sylvie; Bascoul, Céline; Guedeney, Antoine; Delhanty, Patric; Geenen, Vincent; Martens, Henri; Muscatelli, Françoise; Cohen, David; Consoli, Angèle; Payoux, Pierre; Arnaud, Catherine; Salles, Jean-Pierre The Use of Oxytocin to Improve Feeding and Social Skills in Infants With Prader–Willi Syndrome Journal Article PEDIATRICS, 139 (2), pp. e2 0162976, 2017. BibTeX @article{RN10b,
title = {The Use of Oxytocin to Improve Feeding and Social Skills in Infants With Prader–Willi Syndrome},
author = {Maïthé Tauber and Kader Boulanouar and Gwenaelle Diene and Sophie Çabal-Berthoumieu and Virginie Ehlinger and Pascale Fichaux-Bourin and Catherine Molinas and Sandy Faye and Marion Valette and Jeanne Pourrinet and Catie Cessans and Sylvie Viaux-Sauvelon and Céline Bascoul and Antoine Guedeney and Patric Delhanty and Vincent Geenen and Henri Martens and Françoise Muscatelli and David Cohen and Angèle Consoli and Pierre Payoux and Catherine Arnaud and Salles, Jean-Pierre},
year = {2017},
date = {2017-01-01},
journal = {PEDIATRICS},
volume = {139},
number = {2},
pages = {e2 0162976},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
|
Asrir, A; Aloulou, M; Gador, M; Perals, C; Fazilleau, N Interconnected subsets of memory follicular helper T cells have different effector functions Journal Article Nat Commun, 8 (1), pp. 847, 2017, ISSN: 2041-1723 (Electronic) 2041-1723 (Linking). Links | BibTeX @article{RN2b,
title = {Interconnected subsets of memory follicular helper T cells have different effector functions},
author = {Asrir, A. and Aloulou, M. and Gador, M. and Perals, C. and Fazilleau, N.},
url = {https://www.ncbi.nlm.nih.gov/pubmed/29018187},
doi = {10.1038/s41467-017-00843-7},
issn = {2041-1723 (Electronic) 2041-1723 (Linking)},
year = {2017},
date = {2017-01-01},
journal = {Nat Commun},
volume = {8},
number = {1},
pages = {847},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
|
Duguet, F; Locard-Paulet, M; Marcellin, M; Chaoui, K; Bernard, I; Andreoletti, O; Lesourne, R; Burlet-Schiltz, O; Gonzalez de Peredo, A; Saoudi, A Proteomic Analysis of Regulatory T Cells Reveals the Importance of Themis1 in the Control of Their Suppressive Function Journal Article Mol Cell Proteomics, 16 (8), pp. 1416-1432, 2017, ISSN: 1535-9484 (Electronic) 1535-9476 (Linking). Links | BibTeX @article{RN4b,
title = {Proteomic Analysis of Regulatory T Cells Reveals the Importance of Themis1 in the Control of Their Suppressive Function},
author = {Duguet, F. and Locard-Paulet, M. and Marcellin, M. and Chaoui, K. and Bernard, I. and Andreoletti, O. and Lesourne, R. and Burlet-Schiltz, O. and Gonzalez de Peredo, A. and Saoudi, A.},
url = {http://www.ncbi.nlm.nih.gov/pubmed/28373295},
doi = {10.1074/mcp.M116.062745},
issn = {1535-9484 (Electronic) 1535-9476 (Linking)},
year = {2017},
date = {2017-01-01},
journal = {Mol Cell Proteomics},
volume = {16},
number = {8},
pages = {1416-1432},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
|
Laffont, Sophie; Blanquart, Eve; Savignac, Magali; Cenac, Claire; Laverny, Gilles; Metzger, Daniel; Girard, Jean-Philippe; Belz, Gabrielle T; Pelletier, Lucette; Seillet, Cyril; Guery, Jean-Charles Androgen signaling negatively controls group 2 innate lymphoid cells Journal Article J Exp Med, 214 (6), pp. 1581-1592, 2017, ISSN: 1540-9538 (Electronic) 0022-1007 (Linking). Links | BibTeX @article{RN3,
title = {Androgen signaling negatively controls group 2 innate lymphoid cells},
author = {Sophie Laffont and Eve Blanquart and Magali Savignac and Claire Cenac and Gilles Laverny and Daniel Metzger and Jean-Philippe Girard and Gabrielle T Belz and Lucette Pelletier and Cyril Seillet and Jean-Charles Guery},
url = {https://www.ncbi.nlm.nih.gov/pubmed/28484078},
doi = {10.1084/jem.20161807},
issn = {1540-9538 (Electronic) 0022-1007 (Linking)},
year = {2017},
date = {2017-01-01},
journal = {J Exp Med},
volume = {214},
number = {6},
pages = {1581-1592},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
|
Yshii, L M; Hohlfeld, R; Liblau, R S Inflammatory CNS disease caused by immune checkpoint inhibitors: status and perspectives Journal Article Nat Rev Neurol, 13 (12), pp. 755-763, 2017, ISSN: 1759-4766 (Electronic) 1759-4758 (Linking). Links | BibTeX @article{RN5b,
title = {Inflammatory CNS disease caused by immune checkpoint inhibitors: status and perspectives},
author = {Yshii, L. M. and Hohlfeld, R. and Liblau, R. S.},
url = {https://www.ncbi.nlm.nih.gov/pubmed/29104289},
doi = {10.1038/nrneurol.2017.144},
issn = {1759-4766 (Electronic) 1759-4758 (Linking)},
year = {2017},
date = {2017-01-01},
journal = {Nat Rev Neurol},
volume = {13},
number = {12},
pages = {755-763},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
|
Joulia, R; Mailhol, C; Valitutti, S; Didier, A; Espinosa, E Direct monitoring of basophil degranulation by using avidin-based probes Journal Article J Allergy Clin Immunol, 2017, ISSN: 1097-6825 (Electronic) 0091-6749 (Linking). Links | BibTeX @article{RN16,
title = {Direct monitoring of basophil degranulation by using avidin-based probes},
author = {Joulia, R. and Mailhol, C. and Valitutti, S. and Didier, A. and Espinosa, E.},
url = {https://www.ncbi.nlm.nih.gov/pubmed/28433691},
doi = {10.1016/j.jaci.2017.03.030},
issn = {1097-6825 (Electronic) 0091-6749 (Linking)},
year = {2017},
date = {2017-01-01},
journal = {J Allergy Clin Immunol},
keywords = {},
pubstate = {published},
tppubtype = {article}
}
|
